Archive for the ‘Whine Bitch & Me’ Category
老牛吃嫩草
最近，梁志强的“老牛吃嫩草”闹得满成风雨。 报章上也天天在追踪这件事。
我也不想对这件事发表什么。何况我是局外人。
只想对“老牛吃嫩草”， 这句词语发表一下。 “老牛吃嫩草” – 不只是用在男人身上, 也可以用在女人身上哦！=X
Decisions
In life, there are so many decisions to make.
Why why why? No human next life!My BUYS….
I got my Dolly Wink lashes. Lower lashes only. Upper lashes (nice ones) were all SOLD OUT! OMG! 2 pairs at $23.90 and so many people bought? -_-”
I got myself a little piggy too. He looks so pitiful sitting at the shelf. =X
Injury again?
I think I sprained my right wrist! No idea how and when. But I felt the pain this morning already. Damn! Sent from my BlackBerry
Morning movie.
Alice in wonderland. 3D. Sent from my BlackBerry
Ka Ching………… Ka Ching………………
This is what I heard.
After all, it’s my hard work. While people are resting or enjoying, I am busying earning extra money. No particular reasons but I merely don’t want to have my time wasted on unnecessary things/ people. Plus I enjoy the company of kids, they allow to “relax”. Yes, I agree that they do make me angry by not paying attention, not doing homework, but least they are innocent. I feel at ease with them. Least there is no such needs of watching my back while working.Dysmenorrhea
Dysmenorrhea (or dysmenorrhoea) is a medical condition characterized by severe uterine pain during menstruation. While most women experience minor pain during menstruation, dysmenorrhea is diagnosed when the pain is so severe as to limit normal activities, or requiremedication.
Dysmenorrhea can feature differe nt kinds of pain, including sharp, throbbing, dull, nauseating, burning, or shooting pain. Dysmenorrhea may precede menstruation by several days or may accompany it, and it usually subsides as menstruation tapers off. Dysmenorrhea may coexist with excessively heavy blood loss, known as menorrhagia.
Secondary dysmenorrhea is diagnosed when symptoms are attributable to an underlying disease, disorder, or structural abnormality either within or outside the uterus. Primary dysmenorrhea is di agnosed when none of these is detected.
Pathophysiology
During a woman’s menstrual cycle, the endometrium thickens in preparation for potential pregnancy. After ovulation, if the ovum is not fertilized and there is no pregnancy, the built-up uterine tissue is not needed and thus shed.
Molecular compounds calle d prostaglandins are released during menstruation, due to the destruction of the endometrial cells, and the resultant release of their contents.[
1]
Release ofprostaglandins and other inflammatory mediators in the uterus cause the uterus to contract. These substances are thought to be a major factor in primary dysmenorrhea.[2] When the uterine muscles contract, they constrict the blood supply to the tissue of the endometrium, which, in turn, breaks down and dies. These uterine contractions continue as they squeeze the old, dead endometrial tissue through the cervix and out of the body through the vagina. These contractions, and the resulting temporary oxygen deprivation to nearby tissues, are responsible for the pain or “cramps” experienced during menstruation.
Compared with other women, females with primary dysmenorrhea have increased activity of the uterine muscle with increased contractility and increased frequency of contractions
The main symptom of dysmenorrhea is pain concentrated in the lower abdomen, in the umbilical region or the suprapubic region of the abdomen. It is also commonly felt in the right or left abdomen. It may radiate to the thighs and lower back. Other symptoms may include nausea and vomiting, diarrhea or constipation, headache, dizziness, disorientation, hypersensitivity to sound, light, smell and touch, fainting, and fatigue. Symptoms of dysmenorrhea often begin immediately following ovulation and can last until the end of menstruation. This is because dysmenorrhea is often associated with changes in hormonal levels in the body that occur with ovulation. The use of certain types of birth control pills can prevent the symptoms of dysmenorrhea, because the birth control pills stop ovulation from occurring.
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Diagnosis
In one research study using MRI, visible features of the uterus were compared in dysmenorrheic and eumenorrheic (normal) participants. The study concluded that in dysmenorrheic patients, visible features on cycle days 1-3 correlated with the degree of pain, and differed significantly from the control group.[4]
[edit]Treatments
[edit]Nutritional
Several nutritional supplements have been indicated as effective in treating dysmenorrhea, including omega-3 fatty acids, magnesium, vitamin E, zinc, and thiamine (vitamin B1).
Research indicates that one mechanism underlying dysmenorrhea is a disturbed balance between anti-inflammatory , vasodilator eicosanoids derived from omega-3 fatty acids, andproinflammatory, vasoconstrictor eicosanoids derived from  ;omega-6 fatty acids.[5] Several studies have indicated that intake of omega-3 fatty acids can reverse the symptoms of dysmenorrhea, by decreasing the am ount of omega-6 FA in cell membranes.[6] [7][8] The richest dietary source of omega-3 fatty acids is found in flax oil.[9]
Oral intake of magnesium has also been indicated in providing relief: two double-blind, placebo-controlled studies demonstrated a positive therapeutic effect of magnesium on dysmenorrhea.[10] [11] A randomized, double-blind, controlled trial demonstrated that oral intake of vitamin E relieves the pain of primary dysmenorrhea and reduces blood loss.[12] A review of case histories indicated that zinc, in 1 to 3 30-milligram doses given daily for one to four days prior to onset of menses, prevents essentially all to all warning of menses and all menstrual cramping.[13] Intake of thiamine (vitamin B1) was demonstrated to provide “curative” relief in 87% of females experiencing dysmenorrhea, in a controlled study.[14]
[edit]NSAIDs
Non-st eroidal anti-inflammatory drugs (NSAIDs) are effective in relieving the pain of primary dysmenorrhea.[15] NSAIDs can have side effects of nausea, dyspepsia, peptic ulcer, and diarrhea.[16] Patients who cannot take the more common NSAIDs, or for whom they are not effective, may be prescribed a COX-2 inhibitor.[17] One study indicated that conventional therapy with NSAIDs “provides symptomatic relief but has increasing adverse effects with long-term use”,[18] another indicated that long-term use of NSAIDs has “severe adverse effe cts”
Although use of hormonal contraception can improve or relieve symptoms of primary dysmenorrhea,[20][21] a 2001 systematic review found that no conclusions can be made about the efficacy of commonly used modern lower dose combined oral contraceptive pills for primary dysmenorrhea.[22] Norplant[23] and Depo-provera[24][25] are also effective, since these methods often induce amenorrhea. The IntraUterine System (Mirena IUD) has been cited as useful in reducing symptoms of dysmenorrhea.[26] Several non-drug therapies for dysmenorrhea have been studied, including behavioral, acu
Hormonal contraceptives
[edit]Non-drug therapies